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August 01, 1999; 53 (3) Brief Communications

Hereditary ceruloplasmin deficiency increases advanced glycation end products in the brain

K. Tajima, T. Kawanami, R. Nagai, S. Horiuchi, T. Kato
First published August 1, 1999, DOI: https://doi.org/10.1212/WNL.53.3.619
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Article Information

vol. 53 no. 3 619
DOI: 
https://doi.org/10.1212/WNL.53.3.619
PubMed: 
10449130
Published By: 
Wolters Kluwer Health, Inc. on behalf of the American Academy of Neurology
Print ISSN: 
0028-3878
Online ISSN: 
1526-632X
History: 
  • Received January 25, 1999
  • Accepted April 10, 1999
  • First Published August 1, 1999.
Copyright & Usage: 
© 1999

Author Disclosures

    1. K. Tajima, MD, PhD,
    2. T. Kawanami, MD, PhD,
    3. R. Nagai, MD, PhD,
    4. S. Horiuchi, MD, PhD and
    5. T. Kato, MD, PhD
  1. K. Tajima, MD, PhD,
  2. T. Kawanami, MD, PhD,
  3. R. Nagai, MD, PhD,
  4. S. Horiuchi, MD, PhD and
  5. T. Kato, MD, PhD
  1. From the Third Department of Internal Medicine (Drs. TajimaKawanami, and Kato), Yamagata University School of Medicine, Yamagata; and the Department of Biochemistry (Drs. Nagai and Horiuchi), Kumamoto University School of Medicine, Kumamoto, Japan.
  1. Address correspondence and reprint requests to Dr. K. Tajima, Third Department of Internal Medicine, Yamagata University School of Medicine, 2-2-2 Iida-Nishi, Yamagata 990-9585, Japan.
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