Is α-synuclein rising to the surface as a diagnostic biomarker for Parkinson disease?
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Article Information
- First Published October 2, 2013.
Article Versions
- Previous version (October 2, 2013 - 13:00).
- You are viewing the most recent version of this article.
Author Disclosures
- William P. Cheshire, MD and
- Ronald F. Pfeiffer, MD
- William P. Cheshire, MD and
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American Academy of Neurology, Neuro SAE examination writer, 2013
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National Institutes of Health, Autonomic Rare Diseases Clinical Research Consortium, 6102, Principal Investigator, 2011-2013
Mayo Clinic Program in Professionalism & Ethics, PPE-5 grant, Principal Investigator, 2013-2014
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- Ronald F. Pfeiffer, MD
(1) National Parkinson Foundation
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(1)Teva (2)UCB (3)USWorldMeds
(1) Parkinsonism and Related Disorders, Co-Editor in Chief 2008-present (2) Journal of Parkinson's Disease, Editorial Board Member, 2011-present (3) International Journal of Brain Science, Editorial Board Member, 2013-present
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(1) Parkinson's Disease, Second Edition, Taylor & Francis, 2013 (2) Parkinson's Disease and Nonmotor Dysfunction, Second Edition, Humana Press, 2013
(1) University of Tennessee Health Science Center (2) Semmes-Murphey Neurologic & Spine Institute
(1) UCB
(1)Teva (2)UCB (3)USWorldMeds
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(1) Boehringer-Ingelheim (2) UCB (3) Phytopharm
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(1) Columbia University (CORE-PD Study) (2) Weill Cornell Medical College (QE3 Study) (3) Northwestern University (STEADY-PD Study) (4) Indiana University (PROGENI Study)
(1) Parkinson Study Group (2) Michael J Fox Foundation - (I did not receive funding directly from MJFF, but they provided funding for the STEADY-PD Study listed above)
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(1) Tucker Ellis & West, consultant (2) Thomas, Thomas and Hafer, consultant
- From the Department of Neurology (W.P.C.), Mayo Clinic, Jacksonville, FL; and Department of Neurology (R.F.P.), University of Tennessee Health Science Center, Memphis.
- Correspondence to Dr. Cheshire: cheshire{at}mayo.edu
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